Coronary artery hypoxic vasorelaxation is augmented by perivascular adipose tissue through a mechanism involving hydrogen sulfide and cystathionine-β-synthaseTools Donovan, J. and Wong, P.S. and Garle, M.J. and Alexander, S.P.H. and Dunn, W.R. and Ralevic, V. (2018) Coronary artery hypoxic vasorelaxation is augmented by perivascular adipose tissue through a mechanism involving hydrogen sulfide and cystathionine-β-synthase. Acta Physiologica . e13126. ISSN 1748-1708 Full text not available from this repository.AbstractAim: Hypoxia causes vasodilatation of coronary arteries which protects the heart from ischaemic damage through mechanisms including the generation of hydrogen sulfide (H2S), but the influence of the perivascular adipose tissue (PVAT) and myocardium is incompletely understood. This study aimed to determine whether PVAT and the myocardium modulate the coronary artery hypoxic response, and whether this involves hydrogen sulfide.
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